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Clinical Micotoxicosis

Published: January 1, 1900
By: Special Nutrients Inc.
VARIOUS MYCOTOXINS BIOCHEMICAL EFFECTS
(BORRELL 1989)
Aflatoxin* Hepatic size enlargement
Increase in proto-combining time
Pro-ventricle size enlargement
Laying reduction
Embryo development delay
Sericeous protein reduction
Increase in coagulation time
Corporal size reduction
Gizzard size enlargement
Testicle atrophia
Embryo malformations
Antibody absorption reduction
     
Ochratoxin A* Kidney weight increase
Increased Urate deposits
biliary conducts multiplication
Immunization defenses reduction
Corporal Weight loss
Sexual maturity delay
Laying reduction
     
Rubratoxin B* Hepatic hypertrophy
Embryo malformation
Fabricius bag Atrophia
     
Diacetoxycirpernol* Lymphatic tissue necropsy
Biliary vesicle necropsy
Spinal cord necropsy
     
T-2* Mouth necropsy
VII factor and proto-combining reduction
Hepatic hypertrophy
Subcutaneous edema
     
Zearalenone* Liver weight increase
Kidney weight increase
Testicle intestinal fibrosis
Testicle weight reduction

Heart weight increase
Estrogenic effect
Crest size reduction
     
Citrinin* Renal size enlargement
Urine production increase
Increase in water Consumption
     
Penicilloic* Acid Sterigmata-cystine Heart muscle alteration
Hepatic necropsy
Renal necropsy
     
Penitrem A and B* Neurotoxic  
     
Patulin Hepatic alteration  
     
Note: The combination of two or more mycotoxins, even at LOW levels can be more toxic than one toxin at a high level and might NOT show the same symptoms as one mycotoxin.
     
     
SWINE CLINICAL MYCOTOXICOSIS
(OSWEILER, 1992)
Toxin
Category
Clinical Effect
Aflatoxin
Development / grow-out
* Growth and feed conversion reduction.
* Liver microscopic lesions, colangio-hepatitis, hepatic serological enzymes enlargement
* Feed consumption reduction, hardened hair, hypertony.
* Coagulation apathy, depression, anorexia.
* Acute hepatitis, and mortality in 3 to 10 days.
     
  Sow / Reproductive Stage
* No conception effect, normal slow growing suckling pigs because of aflatoxin in the milk.
     
Ochratoxin and Citrinin
Grow-out
* Moderate renal lesions. reduction in weight gain.
* Polydypsia, reduced growth, glycosuria and polyuria
     
  Sows / Reproductive Stage
* Normal when they eat on the first month
     
Tricotecen T-2 and DAS
Development /Grow-out
* Reduced feed consumption, oral and dermal irritation.
* Full rejection and vomiting.
     
Deoxynivalenol
  * Reduced feed consumption.
* Full rejection
     
Zearalenone F-2
Reproductive Sows
* vulvovaginitis, Prolapse
     
  Cycling sows
* Persistency of anestra lutea body.
* Pseudopregnancy
     
  Pregnant sows
* Early embryo mortality when feeding
* 1-3 weeks post-service.
     
Fumonisin
Swine
* Acute lung edema, hepatitis, lymphoblasts genesis paralysis, reduced feed consumption
     
     
VARIOUS MYCOTOXINS BIOCHEMICAL EFFECTS
(SHULLY CHEEKE, 1983)
SYSTEM
MYCOTOXINS
* Metabolic System:
 
- Carbohydrates metabolism
Aflatoxin, Ochratoxin A, Phomopsin A
- Lipids metabolism
Aflatoxin, Ochratoxin A, T-2, Citrinin, Rubratoxin B
- Vitamin assimilation
Aflatoxin, dicumarol
- Protein synthesis
Aflatoxin, Trichothecenes
- Mitochondrial respiration
Aflatoxin, Ochratoxin A, Rubratoxin B, Patulin
* Endocrine system
Aflatoxin, Zearalenone, Ergot, Alkaloids
* Skeletal system
Aflatoxin, Ochratoxin A
   
Note: The combination of two or more mycotoxins, even at LOW levels can be more toxic than one toxin at a high level and might NOT show the same symptoms as one mycotoxin.
   
 
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Dr. Karki Kedar
15 de junio de 2010
hemorrhagic bowel syndrome a new problem we are facing in fattening pigs
Dr. Karki Kedar
20 de mayo de 2008
Thanx for the information. Very informative too. Apart of this, mycotoxin causes encephalomalacia in mule. Dr. Kedar Karki
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