Nutracid Intestim is a new and innovative product for pigs that support growth and improves feed conversion by modulating the immune system, stimulating villi growth and improving absorption capacity.
Under normal physiological conditions most nutrients in feed are directed towards growth. The immune system is only moderately active and therefore requires little energy and nutrients.
During infection, the immune system is triggered and consumes more nutrients, which comes at the expense of the zootechnical performances. According to several researchers, the immune response is often more severe than necessary to eliminate the pathogen. This oversensitivity leads to an inflammatory response which is characterized by an overproduction of pro-inflammatory cytokines. Those signalling molecules target several tissues in the body which eventually results in a lower feed intake, muscle catabolism and fever.
It is therefore important to control the immune response. Anti-inflammatory molecules help to overcome this. They act by inhibiting the overproduction of pro-inflammatory cytokines,thereby shifting the major supply of nutrients from the immune system to growth
Nutracid in the stomach
Nutracid optimizes the pH conditions in the stomach through a partial release of the acids in the stomach. Since there is only a partial release of acids, the development of the endogenous HCL production of the animals will not inhibited, as can be the case with unprotected acids.
Effect of Nutracid in the intestines
The beginning of the small intestine has a very strong buffering capacity, and a strong acidification in the stomach will be neutralized early on in the small intestine. Therefor, addition of unprotected/free acids in the feed will provide only a very limited effect in the intestines.
A progressive release of acids throughout the intestines, as obtained by use of Nutracid products, results in a positive acidification effect in the intestines as well.
Therefore the use of slow-release preparations makes it possible to influence the intestinal microflora, due to the bacteriostatic capacities of organic acids and fatty acids.